The goal of the current study was to answer three primary questions: (a) What are the similarities and differences in the structure and etiology of behavioral disinhibition at the beginning and end of adolescence? (b) Is behavioral disinhibition related to the ability to inhibit prepotent responses? (c) If so, can the relationship between behavioral disinhibition and response inhibition be explained by shared genetic liability? Our results provide clear answers to each of these questions.
