with poorer fibre tract quality across the corpus callosum, internal/external capsules, and posterior corona radiata. The authors interpreted these data to suggest that high-risk drinking (high quantity and/or greater hangover symptoms) may represent an estimate of adverse impact upon WM microstructure. Conversely, hangover symptoms might provide a proxy for difficult to recall estimates of high-risk consumption. No areas of FA were higher for AU versus non-AU youth. These data suggest the potentially damaging effects of infrequent, but high-quantity alcohol exposure on WM integrity and coherence.
