and theta power using a gene-based test and we did not replicate findings at the level of individual SNPs within the gene. Greenwood et al. (2013) carried out a linkage study for antisaccade error in approximately 1000 individuals drawn from families with a schizophrenia proband. They reported linkage to a locus on chromosome 3p14. We examined a 10 Mb region around this locus, testing 39,000 markers for association with antisaccade error, and found no significant results or suggestive evidence that a genetic signal was present (Vaidyanathan, Isen, et al., 2014). One could generate a list of plausible reasons why we could not confirm these Hodgkinson and Greenwood findings in our MTFS sample. However, that would be missing the point. These failures to support previously reported findings represent only two of the many hundreds of leads we pursued in our project, none of which could be confirmed.
