amplitude in AUD [34] and other externalizing disorders [120,121,122]. The heightened impulsivity observed in the AUD group may also be related to altered FC at the frontal connections in the current study and to the lower volumes observed in frontal regions in the same subjects, as reported in our previous study [6]. There is also evidence from other studies that both structural and functional aspects of frontal lobes contribute to increased impulsivity in AUD patients [38,58,123,124]. Furthermore, recent studies have found association of impulsivity with resting state measures of EEG power [125], EEG-based FC [126] and fMRI-based FC [10], suggesting that impulsivity in AUD may underlie specific brain networks.
