Taken together, results suggest that these three endophenotypes are tapping into different neurophysiological and genetic substrates of EXT psychopathology. Results further suggest, however, that some of this genetic variance is common across the endophenotypes. These endophenotypes, then, are making independent, yet overlapping, contributions to the general genetic vulnerability to EXT (particularly in the case of females). This genetic variance that is shared among these measures is the key to their potential utility as a ‘multivariate endophenotype’ for EXT.
