Coheritability analysis showed nominally significant genetic correlations, which were no longer significant after FDR correction. This may be expected due to the relatively low sample size of this GWAS. A nominally significant genetic correlation (r G = .55) was observed between beta oscillations and generalized epilepsy. This is consistent with the putative role of fast beta/gamma oscillations in ictogenesis (in the present sample only nonaffected individuals were used). The largest epilepsy GWAS to date found suggestive evidence for the involvement of GABRA2 (ILAE Consortium, 2014), which we found to be related to beta oscillation power. GABA is a main antiepileptic drug target, and is known to affect (motor) beta EEG via GABAergic modulation of pyramidal cells (Gaetz et al., 2011; Hall et al., 2010; Rowland et al., 2013; Yamawaki, Stanford, Hall, & Woodhall, 2008). We additionally observed a significant genetic correlation between autism and theta oscillations (r G = –.35, p = .009). Although deviant brain function in autism is most consistently found in the lower gamma band due to altered GABA inhibitory neuronal action (Blatt & Fatemi, 2011; Gandal
