studies of rare families with SZ-associated inherited mutations (Lin et al., 2016, Srikanth et al., 2015, Wen et al., 2014, Yoon et al., 2014, Zhao et al., 2015). The ability to precisely modulate SZ disease-relevant gene expression in hiPSC-derived neural cell types would allow hiPSC models to better define the contribution and function of more genes associated with SZ disease risk.
