It is widely assumed that abnormalities of the left temporal lobe (i.e., planum temporale, superior temporal gyrus, hippocampus; e.g., Barta et al., 1990; Bogerts et al., 1990, 1993; Arnold et al., 1991; Shenton et al., 1992; Falkai et al., 1995; Menon et al., 1995; Vita et al., 1995; Pearlson et al., 1997; Kawasaki et al., 2008) are associated with impaired left-lateralized processes typically mediating language-related functions (e.g., Flor-Henry, 1969, 1976; DeLisi et al., 1997; Crow, 2004). Considerable electrophysiologic evidence also suggests that reductions of the classical P3 component, the archetype and most-studied cognitive event-related potential (ERP), in schizophrenia, involve primarily the left side of the brain, which has in turn been attributed to structural temporal lobe abnormalities (e.g., McCarley et al., 1991, 1993, 2002, 2008; O’Donnell et al., 1993, 1999; Egan et al., 1994; Kawasaki et al., 1997; Salisbury et al., 1998; Strik et al., 1994; van der Stelt et al., 2004). Temporal lobe structures are also critically involved in memory formation, storage and retrieval (e.g., Damasio, 1989; Smith and Halgren, 1989), and several neuroimaging studies have linked verbal memory
