severity of AUD (e.g., DSM5AUDSX), and that individuals who successfully reduce their problem drinking might have a less severe form of the disorder, the genetic propensity that extends beyond severity and specifically predicts cessation is distinct. One might speculate that genetic influences on remission relate less to genetic liability to alcohol intake and more to aspects of socio-economic status (e.g., (Trim et al., 2013)), personality characteristics, as well as other comorbid psychiatric disorders (Lopez‐Quintero et al., 2011). Future studies should further explore the heritability of cessation and its co-heritability with other drinking measures.
