Antisocial behaviour (ASB) covers a range of inappropriate behaviours that cause harm to others, the community and the environment. These include aggressive behaviour, hostility, theft, deceitfulness and violent felonies. Apart from the monetary effects(McCollister, French, & Fang, 2010), violent criminal behaviour also has significant social and emotional costs. Communities with high rates of crime often face high unemployment rates and high rates of drug and alcohol abuse, poverty and other social pathologies(Wright, Tibbetts, & Daigle, 2014).Victims of crime, are often left with emotional trauma and can experience serious mental health problems, such as post-traumatic stress disorder(Brewin, Andrews, Rose, & Kirk, 1999). In addition, ASB shows high co-morbidity with other psychiatric traits and maladaptive behaviours(Abram et al., 2015; Goldstein et al., 2017). Against this backdrop, identifying causal mechanisms underlying ASB is critically important to identify prevention and treatment modalities. Accumulated evidence from quantitative and molecular genetic studies reveals the substantial impact of genetic factors in the etiology of ASB. The majority of evidence for a role of genetics is derived from twin studies and, to a lesser extent, adoption studies, and
