for behavior (Palva et al., 2013; Prent & Smit, 2019; Smit et al., 2013) and psychopathology (Linkenkaer‐Hansen, 2005; Montez et al., 2009; Moran et al., 2019; Nikulin et al., 2012). These fast or slow decaying temporal correlations are measurable in EEG, show large individual variation, and are heritable (Linkenkaer‐Hansen et al., 2007). Our goal will be to elucidate how genetic variants affect this oscillatory balance and determine whether these variants are part of excitatory and inhibitory synaptic functioning (such as glutamate and GABA receptor genes). We will investigate whether temporal dynamics vary for participants with a high genetic liability for neurological disorders such as epilepsy, but also for participants with high sensory sensitivity complaints in, for example, autism spectrum disorder (American Psychiatric Association, 2013; Robertson & Baron‐Cohen, 2017) and tinnitus (Hébert et al., 2013).