for enrichment of eQTLs among top signals for coronary artery disease (CAD) or type 2 diabetes (T2D). These results suggest that for at least some complex traits, a more efficient prioritization for replication studies can be achieved by incorporating the expression scores, and that there may be many more SNPs that are truly associated with disease and also likely to be eQTLs than we have identified through GWAS to date.
