EAS and AFR ancestries (RA = 30%; S.E. 7.8% for EAS and RA = 16%; S.E. 3.3% for AFR; Supplementary Fig. 11) below that from participants of SAS ancestry (RA = 37%; S.E. 4.5%). This result is important as it shows how the distribution of SNP effects can lead to unexpected patterns of RA, and therefore challenge straightforward comparisons between traits with different genetic architectures.
