< .0001, ε = 0.9022, and site, F(1, 37) = 5.58, p = .02, were comparable to those found for linked mastoids, but amplitudes were substantially smaller (weak to strong, 5.77 ±3.38, 6.06 ±2.92, 7.33 ±3.01 µV; z vs. Pz, 6. 3 ±3. 2 vs. 5.95 ±2. µV; patients vs. controls, 6.15 ±3. 7 vs. 6.6 ±2. 1 µV). Again, this paralleled the findings for P2 latency, revealing significant main effects of intensity, F(2, 74) = 3.48, p = .05, ε = 0.7982, and site, F(1, 37) = 14.9, p = .0004, but in this case P2 peaked earlier compared to the linked-mastoid data (weak to strong, 781 ±222, 730 ±189, 719 ±192 ms; Cz vs. Pz, 710 ±175 vs. 776 ±222 ms; patients vs. controls, 760 ±220 vs. 726 ±182 ms).
