larger frontal amplitudes for NoGo, in line with a frontal generator for N2 (van Veen and Carter, 2002; Nieuwenhuis et al., 2004). The anteriorly distributed NoGo P3 potentials were also markedly reduced in amplitude in alcoholic subjects as well as in high-risk individuals, indicating impaired inhibitory control in these individuals (Cohen et al., 1997a, b; Kamarajan et al., 2005a, b; Colrain et al., 2011).
