attributable to additive genetic (dotted line) and non-shared environmental (light solid line) factors increased with increasing PSI – however, by jointly modeling PSI with RSI, we were able to capture shared environmental factors that overlap between PSI and RSI – as shown by the dashed line in Panel B (Figure 2), these remained constant across the range of PSI. Panel C (Figure 2) shows standardized results – the relative magnitude of genetic influences in Panel C refer to the proportion of the total variance in RSI attributable to moderated additive genetic factors. As shown here, when standardized, the magnitude of additive genetic (and of non-shared environmental) influence relative to other factors (i.e. C) increases with increasing PSI while the magnitude of shared environmental factors becomes less important with increasing PSI. It is noteworthy that while the effects of C on RSI, all of which are overlapping with PSI, are not moderated, due to moderation of A and E, their relative contribution to the total variance does change with changing PSI. Accordingly, in young women with lower levels of PSI, individual differences in RSI are largely attributable to shared environmental factors – with increasing exposure to substance-using peers, individual differences in
