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Chunk #35 — SUD Prevalence and qEEG Changes — qEEG in Substance Use Disorders — EEG in Cocaine Addiction

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EEG biofeedback as a treatment for substance use disorders: review, rating of efficacy, and recommendations for further research.
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Hans Berger (1937, cited by Gloor 1969; Herning et al. 1985) was the first to study the effects of cocaine on human EEG, reporting an increase in activity in the beta bandwidth. This was replicated in subsequent studies with a larger number of subjects (Alper 1999; Alper et al. 1990, 1998; Costa and Bauer 1997; Herning et al. 1985; Noldy et al. 1994; Prichep et al. 1996, 1999, 2002; Roemer et al. 1995). Beside beta effects, studies have reported an increase in delta activity (Herning et al. 1985) and frontal alpha activity (Herning et al. 1994b), while others have reported an increase in alpha wave EEG associated with bursts of cocaine-induced euphoria (Lukas 1991). More recently, researchers have begun analyzing qEEG profiles of cocaine-dependent patients using the spectral power of each primary bandwidth over the different topographic cortical areas. Excess alpha activity (Alper et al. 1990; Herning et al. 1994b; Lukas 1991; Prichep et al. 1996) and decreased delta activity (Alper et al. 1990; Noldy et al. 1994; Prichep et al. 1996; Roemer et al. 1995) have been reported, while