humans and animals. We predicted that an ethanol-induced increase in the randomness of neuromolecular interactions would result in a reduction of synchrony or phase locking both within a neuronal population and between neuronal populations. To assess this hypothesis, we explored three main questions: (1) Do phase locking measures of EROs differentiate infrequent from frequent tones in rats and humans? (2) Do phase locking measures of EROs differentiate placebo from ethanol in both humans and rats? and (3) Do these measures correlate with a person’s subjective report of intoxication and/or blood ethanol concentration?
