We subsequently fit a model in which CON was entered first. Results indicated that both genetic and environmental influences unique to SR accounted for a significant proportion of the genetic (0.33) and environmental (0.23) covariance between MDD and AUD (Table 4). In this model, 71% of the genetic and 23% of the unique environmental covariation between AUD and MDD was explained by both CON and SR [A = 0.71 (0.42–0.995), E = 0.23 (0.04–0.41)].
