continuous impairment was seen between the two groups).37 On the basis of these findings, the study’s researchers suggested that impairment of episodic memory was the result of the untoward effects of alcohol on the Papez (limbic) circuit and was exacerbated by thiamine deficiency, while the observed impairment in working memory, which was not specific to KS, may have reflected the effects of chronic alcohol consumption on frontocerebellar circuitry.38
