of gene expression levels has frequently been applied to the fine-mapping of risk loci resulting from GWAS, for example in the study of asthma [21] and Crohn’s disease [22]. Furthermore, genome-wide eQTL analysis has proven fruitful in the study of diseases including obesity [23], hypercholesterolemia [24], celiac disease [25], and late-onset Alzheimer disease [26]. In the present study, we have performed a genome-wide screen for eQTLs associated with susceptibility to ALS.
