Alzheimer’s disease is a neurological condition characterised by progressive decline in cognition, with concomitant functional decline.1 The primary pathological hall mark of Alzheimer’s disease is the aggregation of amyloid β peptides into extracellular plaques and of hyperphosphorylated tau into intracellular neurofibrillary tangles, accompanied by neuro inflammation, gliosis, and neurodegeneration.2
