of childhood maltreatment, we cannot establish whether the genetic effect is additive, recessive, or dominant. Across the two cohorts, we found support for recessive effects, additive effects, and to a lesser extent dominant effects. The literature about the interaction between 5-HTTLPR and adversity is limited by the fact that most studies to date have only reported a test of one genetic model and have not systematically compared different genetic models. As such, extant research does not, at present, support any genetic model above others (Caspi et al., 2010). We hope that researchers will be encouraged by the present report to provide full information about different genetic models, allowing transparent interrogation of accumulating findings.
