affected the findings. The alternative, coding them as not meeting criteria for the disorder, would have added solely to those cells containing individuals without PTSD and each type of trauma exposure, thus increasing the magnitude of correlations between each trauma type and PTSD and the respective estimates of their total shared variance. The present approach reduced the number of comparisons informative for calculating parameter estimates for PTSD and, thus, limited our ability to examine alternative models (eg, obtaining separate estimates for women and men). Eighth, given the sample’s complex family structure and sample size constraints, we used models that controlled for the main effect of sex. By doing so, we may have failed to detect underlying genotype × sex interactions.
