Etiology of early age onset substance use disorder: a maturational perspective.
paper
Cited
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- Authors
- Tarter, R; Vanyukov, M; Giancola, P; Dawes, M; Blackson, T; Mezzich, A; Clark, D B
- Year
- 1999
- Journal
- Development and psychopathology
- PMID
- 10624720
- DOI
- 10.1017/s0954579499002266
The etiology of early age onset substance use disorder (SUD), an Axis I psychiatric illness, is examined from the perspective of the multifactorial model of complex disorders. Beginning at conception, genetic and environment interactions produce a sequence of biobehavioral phenotypes during development which bias the ontogenetic pathway toward SUD. One pathway to SUD is theorized to emanate from a deviation in somatic and neurological maturation, which, in the context of adverse environments, predisposes to affective and behavioral dysregulation as the cardinal SUD liability-contributing phenotype. Dysregulation progresses via epigenesis from difficult temperament in infancy to conduct problems in childhood to substance use by early adolescence and to severe SUD by young adulthood.
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