GNAI gene

Aliases

Gi, Gi/o, Gαi/o

External links

NCBI Gene · Ensembl · GeneCards · UniProt

Related entities (5)

Mentioned in (13)

Papers in which this entity is mentioned.

• Cannabinoids Exacerbate Alcohol Teratogenesis by a CB1-Hedgehog Interaction. (2019)
• Smoothened determines β-arrestin-mediated removal of the G protein-coupled receptor Gpr161 from the primary cilium. (2016)
• CB1 cannabinoid receptor-mediated increases in cyclic AMP accumulation are correlated with reduced Gi/o function. (2016)
• Cognitive Impairment Induced by Delta9-tetrahydrocannabinol Occurs through Heteromers between Cannabinoid CB1 and Serotonin 5-HT2A Receptors. (2015)
• Gene × environment interaction studies have not properly controlled for potential confounders: the problem and the (simple) solution. (2014)
• AT1R-CB₁R heteromerization reveals a new mechanism for the pathogenic properties of angiotensin II. (2011)
• Sonic hedgehog signaling is decoded by calcium spike activity in the developing spinal cord. (2011)
• Molecular assays for characterization of alternatively spliced isoforms of the u opioid receptor (MOR). (2010)
• Brain stress systems in the amygdala and addiction. (2009)
• Protein kinases and addiction. (2008)
• Activation of heterotrimeric G proteins by Smoothened. (2006)
• In utero cocaine-induced dysfunction of dopamine D1 receptor signaling and abnormal differentiation of cerebral cortical neurons. (2000)
• Concurrent stimulation of cannabinoid CB1 and dopamine D2 receptors augments cAMP accumulation in striatal neurons: evidence for a Gs linkage to the CB1 receptor. (1997)

Merged raw entities (3)

All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.