coronary artery disease phenotype
Evidence from:
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Mentioned in (17)
Papers in which this entity is mentioned.
- Modeling heterogeneity in single-cell perturbation states enhances detection of response eQTLs. (2025)
- Concordance between male- and female-specific GWAS results helps define underlying genetic architecture of complex traits. (2025)
- Integrative polygenic risk score improves the prediction accuracy of complex traits and diseases. (2024)
- Recent advances in polygenic scores: translation, equitability, methods and FAIR tools. (2024)
- Biobank-scale methods and projections for sparse polygenic prediction from machine learning. (2023)
- Polygenic scoring accuracy varies across the genetic ancestry continuum. (2023)
- Polygenic risk scores: from research tools to clinical instruments. (2020)
- ASEP: Gene-based detection of allele-specific expression across individuals in a population by RNA sequencing. (2020)
- Cancer PRSweb: An Online Repository with Polygenic Risk Scores for Major Cancer Traits and Their Evaluation in Two Independent Biobanks. (2020)
- Polygenic prediction via Bayesian regression and continuous shrinkage priors. (2019)
- Integrating predicted transcriptome from multiple tissues improves association detection. (2019)
- Integrating predicted transcriptome from multiple tissues improves association detection. (2019)
- Genome-wide association study of alcohol consumption and use disorder in 274,424 individuals from multiple populations. (2019)
- The 3D Genome Browser: a web-based browser for visualizing 3D genome organization and long-range chromatin interactions. (2018)
- Genome-wide Association Study of Cannabis Dependence Severity, Novel Risk Variants, and Shared Genetic Risks. (2016)
- A gene-based association method for mapping traits using reference transcriptome data. (2015)
- 3' end mRNA processing: molecular mechanisms and implications for health and disease. (2008)
Merged raw entities (1)
All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.
| Raw name | Type | Papers | Mentions |
|---|---|---|---|
| coronary artery disease | phenotype | 44 | 93 |