Bax gene
Evidence from:
primary |
all sources
Related entities (1)
| Subject | Relation | Object | p-value | Evidence |
|---|---|---|---|---|
| miRNA | regulates | Bax | — | 1 |
Mentioned in (29)
Papers in which this entity is mentioned.
- The pro-apoptotic Bax gene modifies susceptibility to craniofacial dysmorphology following gastrulation-stage alcohol exposure. (2022)
- Transcriptomic analyses of gastrulation-stage mouse embryos with differential susceptibility to alcohol. (2021)
- Loss of tumor protein 53 protects against alcohol-induced facial malformations in mice and zebrafish. (2021)
- Transcriptome-Wide Regulation of Key Developmental Pathways in the Mouse Neural Tube by Prenatal Alcohol Exposure. (2020)
- Murine Models for the Study of Fetal Alcohol Spectrum Disorders: An Overview. (2020)
- CHASMplus Reveals the Scope of Somatic Missense Mutations Driving Human Cancers. (2019)
- Designing combination therapies with modeling chaperoned machine learning. (2019)
- Epigenetic mechanisms in alcohol- and adversity-induced developmental origins of neurobehavioral functioning. (2018)
- Effects of prenatal alcohol exposure (PAE): insights into FASD using mouse models of PAE. (2018)
- Knockdown of Mns1 Increases Susceptibility to Craniofacial Defects Following Gastrulation-Stage Alcohol Exposure in Mice. (2018)
- A Next Generation Connectivity Map: L1000 Platform and the First 1,000,000 Profiles. (2017)
- The Genetics of Fetal Alcohol Spectrum Disorders. (2016)
- Establishment of human iPSC-based models for the study and targeting of glioma initiating cells. (2016)
- Fetal Alcohol Spectrum Disorders: An Overview from the Glia Perspective. (2015)
- Genetic absence of nNOS worsens fetal alcohol effects in mice. II: microencephaly and neuronal losses. (2015)
- Importance of genetics in fetal alcohol effects: null mutation of the nNOS gene worsens alcohol-induced cerebellar neuronal losses and behavioral deficits. (2015)
- MiR-125b protects against ethanol-induced apoptosis in neural crest cells and mouse embryos by targeting Bak 1 and PUMA. (2015)
- Low-dose maternal alcohol consumption: effects in the hearts of offspring in early life and adulthood. (2014)
- Long-term genomic and epigenomic dysregulation as a consequence of prenatal alcohol exposure: a model for fetal alcohol spectrum disorders. (2014)
- Differential expression profiles of glycosphingolipids in human breast cancer stem cells vs. cancer non-stem cells. (2013)
- Alcohol-induced apoptosis of oligodendrocytes in the fetal macaque brain. (2013)
- Early exposure to alcohol leads to permanent impairment of dendritic excitability in neocortical pyramidal neurons. (2012)
- Enhanced deficits in long-term potentiation in the adult dentate gyrus with 2nd trimester ethanol consumption. (2012)
- Protection of neurons and microglia against ethanol in a mouse model of fetal alcohol spectrum disorders by peroxisome proliferator-activated receptor-γ agonists. (2011)
- The role of zinc in the modulation of neuronal proliferation and apoptosis. (2010)
- The epigenetics of sex differences in the brain. (2009)
- Modeling the impact of alcohol on cortical development in a dish: strategies from mapping neural stem cell fate. (2008)
- Nicotinamide protects against ethanol-induced apoptotic neurodegeneration in the developing mouse brain. (2006)
- Insulin-like growth factor-I overexpression attenuates cerebellar apoptosis by altering the expression of Bcl family proteins in a developmentally specific manner. (2001)
Merged raw entities (2)
All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.
| Raw name | Type | Papers | Mentions |
|---|---|---|---|
| bax | gene | 29 | 122 |
| bcl2-associated x protein | gene | — | — |