open field phenotype
Evidence from:
primary |
all sources
Related entities (2)
| Subject | Relation | Object | p-value | Evidence |
|---|---|---|---|---|
| dipoles | associated_with | open field | — | 1 |
| scalp | associated_with | open field | — | 1 |
Mentioned in (12)
Papers in which this entity is mentioned.
- Prenatal alcohol exposure disrupts Sonic hedgehog pathway and primary cilia genes in the mouse neural tube. (2021)
- The enduring impact of neurulation stage alcohol exposure: A combined behavioral and structural neuroimaging study in adult male and female C57BL/6J mice. (2018)
- Acute alcohol exposure during neurulation: Behavioral and brain structural consequences in adolescent C57BL/6J mice. (2016)
- Importance of genetics in fetal alcohol effects: null mutation of the nNOS gene worsens alcohol-induced cerebellar neuronal losses and behavioral deficits. (2015)
- LRP1 is critical for the surface distribution and internalization of the NR2B NMDA receptor subtype. (2013)
- Generator localization by current source density (CSD): implications of volume conduction and field closure at intracranial and scalp resolutions. (2012)
- Maternal immune activation yields offspring displaying mouse versions of the three core symptoms of autism. (2012)
- Adolescent binge drinking alters adult brain neurotransmitter gene expression, behavior, brain regional volumes, and neurochemistry in mice. (2011)
- Fetal alcohol exposure leads to abnormal olfactory bulb development and impaired odor discrimination in adult mice. (2011)
- The effects of a single memantine treatment on behavioral alterations associated with binge alcohol exposure in neonatal rats. (2011)
- Prenatal alcohol exposure and chronic mild stress differentially alter depressive- and anxiety-like behaviors in male and female offspring. (2010)
- Maternal immune activation alters fetal brain development through interleukin-6. (2007)
Merged raw entities (1)
All extracted name/type variants the normalize job merged into this entity. Use this to spot wrong merges, or aliases that should be split off.
| Raw name | Type | Papers | Mentions |
|---|---|---|---|
| open field | phenotype | 12 | 18 |