Ctbp2 regulates EMT
Evidence from:
primary |
all sources
Evidence (6 sources)
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
CtBP2 ... promoted the EMT phenotype of HCC cells.
confidence: 0.95
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
CtBP2 overexpression promoted cell motility and invasion by inducing EMT in HCC cells.
confidence: 0.95
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
CtBP2 bound SNAI1 as a transcriptional co-repressor and, thereby, promoted GLI1/SNAI1 driven EMT in HCC.
confidence: 0.90
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
CtBP2 promotes HCC cell mobility and invasion by inducing EMT
confidence: 0.93
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
CtBP2 ... contributes to inducing the EMT phenotype
confidence: 0.95
CtBP2 is an independent prognostic marker that promotes GLI1 induced epithelial-mesenchymal transition in hepatocellular carcinoma.
(2015)
PMID:25686837
cited
SNAI1 was required for CtBP2-driven EMT
confidence: 0.80