ethanol group regulates Gabra1
Evidence from:
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Evidence (9 sources)
chronic EtOH exposure selectively increases the internalization of α1 GABAA receptors ... There is also a decrease in α1 GABAA receptors
confidence: 0.85
decrease in α1 GABAA receptors ... following chronic EtOH exposure
confidence: 0.95
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
Chronic ethanol exposure selectively increases the internalization of α1‑GABAA receptors ... of the cerebral cortex.
confidence: 0.95
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
Receptor cross-talk plays a vital role in modulation of GABAA receptor function and expression following ethanol exposure.
confidence: 0.85
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
Ethanol exposure increases tyrosine kinase phosphorylation of the GABAA receptor subunits α1, β2, and γ2.
confidence: 0.90
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
Chronic ethanol administration ... reduces ... GABAA receptor α1 ... mRNA/peptide levels in cerebral cortex
confidence: 0.92
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
similar to the effects of ethanol on GABAA receptor α1 subunit expression
confidence: 0.92
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
α1 subunit peptide and mRNA are decreased following chronic ethanol administration in cerebellum
confidence: 0.90
The role of GABA(A) receptors in the acute and chronic effects of ethanol: a decade of progress.
(2009)
PMID:19455309
cited
Chronic ethanol consumption decreases expression of the α1 subunit at the cell surface
confidence: 0.93