Kcnj6 risk_factor_for short-term potentiation
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Evidence (1 sources)
Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome.
(2017)
PMID:28342823
cited
Kir3.2 subunit-containing potassium channels contribute to defects in synaptic plasticity in Ts65Dn mice.
confidence: 0.85