of GFAP and levels of IL1B or IL6 (Supplementary Figures 5a and b). The expression levels of the two inflammation marker genes showed significant correlation (Supplementary Figure 5c). The RIN and pH of schizophrenia samples were significantly and marginally lower than those of controls, respectively (Supplementary Table 1). However, the RIN (pH) and GFAP expression levels in the postmortem brain samples were not significantly correlated to each other: P=0.706 (0.987), 0.585 (0.919) and 0.120 (0.682) for control, schizophrenia and total brains, respectively. Therefore, the results suggest an elevated gliogenic competence in schizophrenia without 22q11.2 deletion.
