was present in all PDGFRα+‐OPCs (iPSC91/iPSC92, n = 5/5, N = 1/1), but not oligodendrocytes (iPSC91/iPSC92, n = 5/4, N = 1/1). In addition, an equivalent reduction in AMPAR unitary conductance (Fig. 7H) as they mature from OPCs to oligodendrocytes are seen in the iPSC9 lines compared with control lines. Collectively these findings are consistent with an absence of maturational deficit in mutant C9ORF72‐derived oligodendrocyte‐lineage cells.
