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Chunk #106 — Clinical Considerations: Comorbidities of SUD and Implications for Individualized (qEEG-Guided) Neurofeedback

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EEG biofeedback as a treatment for substance use disorders: review, rating of efficacy, and recommendations for further research.
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It may make good sense clinically to consider specific neurotherapy treatment of these disorders either in place of or preceding alpha-theta therapy, similar to the Scott–Kaiser approach. Second, applicable neurotherapy approaches are attractive alternative therapies for coexisting or underlying conditions in SUD clients who have high-risk behaviors for medication treatment, such as overdosing, abuse, or poor compliance. While there are no published systematic studies of neurotherapy treatment of co-occurring depression, TBI, ADHD, PTSD, or drug neurotoxicity on the course and outcome of addictive disorders, several recent reports of neurotherapy for addictions based on qEEG findings, which in turn may be related to comorbidities, have been presented. Basically, this technique involves the use of qEEG to identify patterns of EEG that deviate from standardized norms, and individualized EEG biofeedback protocols to correct them (Romano-Micha 2003). DeBeus et al. (2002) are presently conducting a randomized controlled study of neurotherapy for SUD that examines the difference between a qEEG-based treatment, a research-based (Scott–Peniston) treatment, and a wait-list control for chemically dependent outpatients. Preliminary results are promising. While historically, alpha-theta training has been the