conducting a randomized controlled study of neurotherapy for SUD that examines the difference between a qEEG-based treatment, a research-based (Scott–Peniston) treatment, and a wait-list control for chemically dependent outpatients. Preliminary results are promising. While historically, alpha-theta training has been the accepted approach in treating chemical dependency, this study suggests qEEG-based training is a viable alternative, demonstrating similar outcomes for personality change and abstinence rates. Future directions include determination of those likely to benefit from one of the particular treatments or a combination of the two and analysis of long-term abstinence rates. Gurnee (2004) has presented data on a series of 100 sequential participants with SUD who were treated by qEEG-based neurotherapy, with marked heterogeneity of qEEG subtypes and corresponding symptom complexes. In this clinically derived scheme, qEEGs that deviate from normative databases, mainly with excess alpha amplitude, are associated more often with depression and ADD. Those with deficient alpha amplitude are associated with anxiety, insomnia, and alcohol/drug abuse. Beta excess amplitude is associated with anxiety, insomnia, and alcohol/drug abuse. Central abnormalities are interpreted as mesial frontal dysfunction and are associated with anxiety, rumination, and obsessive compulsive symptoms. The therapeutic approach is to base neurotherapy on correcting identified qEEG abnormalities, i.e.,
