As should be clear by now, the degree to which the individual phenotypes reflect different sets of risk factors is a function of the magnitude of the different paths. For example, unlike the above situation, if paths a11 and a12, and paths a21 and a22 are similar in magnitude, then EP1 and EP2 will be substantially correlated and they will be nonspecific, indexing in a similar manner the first and second genetic factor. A model analogous to Figure 5 could be developed that assumed a mediational model for EPs. It would be considerably more complex and will not be presented here.
