As mentioned above, α oscillations also play a role in long-range neural communication and resting-state α power correlates positively with DMN activity (Mantini et al., 2007; Jann et al., 2009; Sadaghiani et al., 2010; Knyazev et al., 2011; Mo et al., 2013; Samogin et al., 2019). The α deficits in DMN hubs thus dovetails the extant literature citing both deficient α activity (Clancy et al., 2017; Clancy et al., 2020) and DMN hypoactivity in PTSD (Koch et al., 2016; Akiki et al., 2018). Notably, resting-state β and θ oscillations have also been found to be associated with DMN activity (Laufs et al., 2003; Mantini et al., 2007; Scheeringa et al., 2012). However, prior sensor-level analyses have not revealed PTSD-related anomalies in the β or θ frequencies (ps > 0.16; Clancy et al., 2017). Future research is warranted to examine the other oscillatory activities in various states or tasks to elucidate their contribution to PTSD pathology.
