The increased beta1 EEG-SFC in the SCZ group is mainly seen in the posterior regions of the right hemisphere; SDD showed greater EEG-SFC specifically between left temporal, left orbitofrontal, and right anterior cingulate ROI pairs. When compared separately against HV, LDD, and SDD showed different patterns of increased EEG-SFC. An increase of beta1 EEG-SFC was recently described in patients with at-risk mental state (ARMS) for psychosis, suggesting that atypical beta-band source synchronization could be an indicator of an increased vulnerability for SCZ/psychosis spectrum (Ramyead et al., 2014). Furthermore, patients with short disease duration showed greater EEG-SFC in the beta2 band compared with those with longer duration who show widespread reduction in EEG-SFC compared with HV. Although results of beta2 FC do not appear as univocal as those of the other frequency bands, they could be explained by hypothesizing that the initial pattern of hyperconnectivity, which has indeed been demonstrated in paradigm-related MEG connectivity studies (Sun et al., 2013), could in time be followed by the diffuse deficit seen in chronic patients. Comparison with other EEG studies (see for review Boutros
