Alcohol use disorder (AUD) is characterized by a loss of executive control over the timing and extent of alcohol consumption that is concretely manifests as an inability to moderate alcohol craving, seeking, and ingesting despite adverse consequences (Ghin et al., 2022). Investigations into the neurobiological mechanisms underlying AUD and other substance abuse disorders point to dysregulation in brain regions that mediate decision making, planning, and reward (Koob & Volkow, 2016). There has been a growing body of evidence indicating that compromised functional connectivity (FC) contributes to the dysregulation in these brain networks (Kinreich, McCutcheon, et al., 2021; Meyers et al., 2021; Song et al., 2024). Conceptually, FC refers to a statistical measure of coordination of neural activity between different brain regions across time, and several methods for extracting FC metrics from functional magnetic resonance imaging (fMRI), electroencephalography (EEG), and magnetoencephalography (MEG) data have been developed (Constable et al., 2013; Wang et al., 2014). AUD-associated changes to FC have been reported using methods such as fMRI seed-based connectivity analyses and EEG coherence (Kamarajan et al., 2020; Kinreich, McCutcheon, et al., 2021;
