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Chunk #46 — Results — Loss of nFGFR1 signaling affects developmental genome programing

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Cerebral organoids reveal early cortical maldevelopment in schizophrenia-computational anatomy and genomics, role of FGFR1.
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NCCNPC vs. NPC+ FGFR1(SP-/NLS)(TK-)NCC+ vs. NCC+ FGFR1(SP-/NLS)(TK-)NCC vs. NCC+ FGFR1(SP-/NLS)Neural development Synapse development, assemblyYes Synaptic plasticity NS/CNS developmentYesYes Brain parts developmentYesYesYes Forebrain developmentYes Cell proliferation, mitotic cycleYesYesYes Cell migrationYesYes NeurogenesisYesYesYes Cell adhesionYes Extracellular matrix, proteasesYesYes Cell, neuronal differentiationYesYesYes Axon developmentYes Axonal guidanceYesOligodendrocyte development Glial development Neurotransmission Neuronal projectionYes Learning and MemoryResponse to retinoic acid2nd messenger signalingRegulation of signal transductionRegulation of signal transductionCell–cell signaling Apoptosis, neuron apoptosisYesYesYesDevelopment general Organ and developmental morphogenesisYesYes AngiogenesisYes Cardiovascular developmentYes Multicellular organism developmentYesYesYes Cellular response to TGFβ Limb, organ morphogenesisYesYes Eye developmentYesYesDevelopmental inductionGene expressionTranscription RNA PolIITranscription RNA PolIICategories affected only in FGFR1(SP-/NLS)(TK-) or FGFR1(SP-/NLS) transfected cells are listed by names