reduced olfactory function in schizophrenia is not secondary to deficits in attention or executive function (Seidman et al., 1997). It still remains to be demonstrated, however, whether the OERP deficits in schizophrenia are specific to olfactory processing or stem from a frontotemporal dysfunction that affects ERPs in multiple modalities. Given our N2-like interpretation of the olfactory N1 sink, its marked reduction in schizophrenia is in striking accordance with ERP evidence documenting profound reductions of N2 amplitudes across processing modalities and paradigms (e.g., Alain, Bernstein, He, Cortese, & Zipursky, 2002; Alain, Cortese, Bernstein, He, & Zipursky, 2001; Bruder et al., 1998, 1999; Kayser et al., 1999, 2001, 2009; O’Donnell et al., 1993; Umbricht, Bates, Lieberman, Kane, & Javitt, 2006).
