For psychiatric-immune phenotype-pairs that demonstrated a significant genome-wide correlation with the LDSC method (i.e., those in Table 1), we used the HESS software to examine the genetic correlation within the ~1694 partitioned genomic loci. The number of correlated loci before and after BH multiple test correction are depicted in Table 3; detailed results for these analyses, including local heritability, correlation strength, and the lists of gene symbols within each loci are provided in Supplementary Table 3. Only SZ displayed robust local genetic correlations with immune-related phenotypes, including thirty-two loci with CD, 37 loci with PBC, 20 loci with SLE, and 8 with UC (Table 3, depicted in Figure 2). Upon closer examination of the loci implicated between SZ and CD, we noticed that five of these loci contained GW hits, including one locus on chromosome 4q24 (4:100678360-103221356; highlighted green in Figure 2) that contained GW hits for both SZ and CD within the present data, and with 4 other autoimmune diseases (immunobase.org); these signals are near autoimmunity candidate genes NFKB1 and MANBA, as well as proposed SZ candidate gene SLC39A8,
