bipolar patients (Clementz et al., 1994) and even more reliably in patients with schizophrenia (Begic et al., 2011; Clementz et al., 1994; Harris, Melkonian, Williams, & Gordon, 2006; Sponheim, Clementz, Iacono, & Beiser, 1994, 2000; Sponheim, Iacono, Thuras, Nugent, & Beiser, 2003). However, EEG abnormalities are more often reported for schizophrenic patients than their unaffected relatives, suggesting that they may reflect disease status, illness progression, or long-term medication effects (Ranlund et al., 2014; Venables, Bernat, & Sponheim, 2009), rather than endophenotypic properties.
