Changes in EEG activity have also been reported in patients with neurodegenerative conditions, such as Alzheimer’s disease, which is characterized by a slowing of the dominant (alpha) frequency as well as increases in low-frequency power (Petit, Gagnon, Fantini, Ferini-Strambi, & Montplaisir, 2004; Stam, 2005). The anomalies in this case appear to reflect the disease process rather than a genetically influenced vulnerability. However, understanding the molecular-genetic influences on the EEG parameters themselves is relevant.
