was 138% (1.2 drinks/wk) higher for women and 99% (2.7 drinks/wk) higher for men, compared to those with older onset. Among those with middle onset, PYDD was found to be 55% (0.5 drinks/wk) higher among women and 70% (1.3 drinks/wk) higher among men for each additional event (up to 3), when compared to those with older onset. Given the distribution of our SLE variables, we coded totSLE as 0, 1, 2, 3+ and used binary coding (0 vs. 1+) for iSLE and dSLE. This difference from Dawson and colleagues’ (2007) coding of SLE (0, 1, 2, 3 to 5, 6+) is likely to have contributed to differences in our findings. By restricting our analyses to SLE with at least high-moderate long-term threat, our sample had fewer SLE (0.7 totSLE vs. 2 in the NESARC study). Our intent was to exclude minor events that would not be expected to be influence drinking as measured over the course of a year, but this may also have reduced our power to detect some interactions in subsets of the data.
