One goal of examining gene regulation in the brain is to inform the analyses of genes that may influence risk for alcoholism. Toward that end, we compiled data from 10 previously published gene expression studies (Flatscher-Bader et al., 2010; Flatscher-Bader et al., 2005; Iwamoto et al., 2004; Kryger & Wilce, 2010; Lewohl et al., 2000; Liu et al., 2007; Liu et al., 2006; Mayfield et al., 2002; Sokolov et al., 2003; Zhou et al., 2011b), from this study, and from 12 GWAS for risk of alcoholism or alcoholic traits (Bierut et al., 2010; Edenberg et al., 2010; Foroud et al., 2007; Hack et al., 2011; Johnson et al., 2011; Kendler et al., 2011; Lind et al., 2010; Treutlein et al., 2009; Wang et al., 2012; Xuei et al., 2006; Zlojutro et al., 2011; Zuo et al., 2012). There were 386 genes identified by at least 2 of these collected studies (Supplemental Table S4). Five genes were identified in 4 studies, and are thus strong candidates for further study: selenoprotein P (SEPP1), heterochromatin protein 1 binding protein 3 (HP1BP3), transferrin (TF),
