and sinks were fewer and weaker in alcoholics. In a sample of individuals at high risk (HR) for alcoholism during the performance of an auditory oddball task, Ramachandran et al. (1996) reported significant reductions in both P3 amplitudes and P3-related CSD activations in the HR group over the posterior central, parietal, and occipital areas, along with prominent topographic differences in the CSD maps. Similarly, in a three stimuli auditory oddball paradigm, Hada et al. (2001) found that the HR group manifested significantly lower P3a amplitudes as well as a highly differentiated CSD map characterized by more but weaker posterior sources for P3a compared to low-risk subjects.
