the shared environmental correlations that were also constrained to equality. The genetic correlations were estimated at +0.78 and the shared environmental correlations at +0.80. However, this model did not fit quite as well as the first model. Our third model constrained to equality the common factor genetic and environmental sex correlations (rac = rcc), and the specific genetic and environmental sex correlations (ras = rcs). The two common and syndrome-specific factor correlations were estimated at +0.79 and +0.80, respectively. But again this model fit modestly worse than model 1. Our fourth model constrained the common factor genetic and environmental correlations to unity (rac = rcc = 1), and constrained the specific genetic and environmental correlations to equality separately for DA, CB and AUD. The model estimated the between sex genetic and environmental correlations for DA and AUD to be high (+.92 and +0.98, respectively) but the estimated correlation for CB was lower (+0.37). However, this model also fitted worse than model 1.
