Our model had four major genetic or shared environmental correlations between the sexes that could be estimated (rac, ras, rcc, and rcs) where correlations related to the specific syndromes (ras and rcs) could be the same or differ between DA, CB and AUD. However, a model with separate estimates of all correlations was not identified and therefore, of the very large number of possible of models, we chose on a priori grounds to examine four. As seen in Table 5, our first and simplest common pathway model constrained to unity all four relevant sex correlations: rac =ras = rcc = rcs = 1. The second model gave us optimal power to distinguish the genetic and environmental correlations for the externalizing disorders in males and females. We set all the genetic correlations to equality and allowed them to differ from all the shared environmental correlations that were also constrained to equality. The genetic correlations were estimated at +0.78 and the shared environmental correlations at +0.80. However, this model did not fit quite as well as the first model. Our third model
